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http://krishi.icar.gov.in/jspui/handle/123456789/50539| Title: | Virus induced gene silencing of Xa38 compromises the resistance towards bacteria blight of rice. |
| Other Titles: | Not Available |
| Authors: | Kant R, Kumari K, Mishra P, Rai R, Singh K and Dasgupta I |
| ICAR Data Use Licennce: | http://krishi.icar.gov.in/PDF/ICAR_Data_Use_Licence.pdf |
| Author's Affiliated institute: | ICAR::National Institute for Plant Biotechnology |
| Published/ Complete Date: | 2021-01-01 |
| Project Code: | Not Available |
| Keywords: | VIGS, bacterial blight, rice, Xa38 |
| Publisher: | Not Available |
| Citation: | Not Available |
| Series/Report no.: | Not Available; |
| Abstract/Description: | Bacterial blight (BB) is one of the most destructive diseases of rice, caused by the bacterial pathogen Xanthomonas oryzae pv. oryzae (Xoo). Several BB resistant genes have been deployed in rice cultivars. One such gene is Xa38, which exhibits resistance against several Indian Xoo races. Since the detailed characterization of Xa38 is still awaited, we have used Virus Induced Gene Silencing, using the rice-specific pRTBV-MVIGS system, to silence Xa38 and study its effect on the symptom development of BB. We find that Xa38 silencing compromises the resistance towards Xoo. Silencing results in increased BB symptoms and enhanced Xoo accumulation |
| Description: | Not Available |
| ISSN: | Not Available |
| Type(s) of content: | Research Paper |
| Sponsors: | Not Available |
| Language: | English |
| Name of Journal: | Physiological and Molecular Plant Pathology |
| NAAS Rating: | 7.65 |
| Impact Factor: | 2.7 |
| Volume No.: | Not Available |
| Page Number: | Not Available |
| Name of the Division/Regional Station: | Not Available |
| Source, DOI or any other URL: | Not Available |
| URI: | http://krishi.icar.gov.in/jspui/handle/123456789/50539 |
| Appears in Collections: | CS-NIPB-Publication |
Files in This Item:
| File | Description | Size | Format | |
|---|---|---|---|---|
| PMPP-Xa38 paper_2021.pdf | 3.33 MB | Adobe PDF | View/Open |
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