Dynamics of anthracnose disease causing pathogen and inheritance and ssr marker- assisted tagging of resistance to anthracnose in chilli
KrishiKosh
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Title |
Dynamics of anthracnose disease causing pathogen and inheritance and ssr marker- assisted tagging of resistance to anthracnose in chilli
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Creator |
C, Nanda
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Contributor |
A, Mohan Rao
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Subject |
fruits, biological phenomena, diseases, fungi, chillies, genetics, planting, yields, cytoplasm, crossing over
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Description |
Genetic options are considered as the most economical and sustainable strategies to reduce losses due to anthracnose disease in chilli. An attempt was made to understand the dynamics of host-pathogen interaction, inheritance of anthracnose resistance and to identify DNA markers linked to genomic regions controlling chilli anthracnose resistance at the Hot Pepper Improvement Unit, Department of Genetics and Plant Breeding, University of Agricultural Sciences, GKVK, Bengaluru during 2006-2010. Anthracnose disease in chilli was prevalent in all the surveyed districts of Karnataka and Andhra Pradesh. Colletotrichum spp. isolates were identified unambiguously as Colletotrichum capsici (Cc) and Colletotrichum gloeosporioides (Cg) based on conidial shape. DNA-based assay using ITS and AFLP markers supplemented morphological trait-based assay for delineating species identity of Colletotrichum isolates. Both Cg and Cc were equally frequent in the surveyed chilli growing regions. The isolates ‘Cc 38’ and ‘Cg 33’ sampled from Khammam district of Andhra Pradesh were found most virulent. The SSR marker HpmsE081 was found significantly linked with genomic regions controlling resistance to anthracnose caused by ‘Cc 38’. However, the association appeared to be very weak as suggested by low contribution of marker variance coupled with low heritability. Fewer to large number of genes, respectively were involved in controlling resistance to anthracnose caused by ‘Cc 38’ and ‘Cg 33’. Fewer numbers of genes with decreasing effects and complementary gene interactions appeared to control the inheritance of number of fruits per plant and fruit length. Large number of genes with decreasing effects and complementary gene interactions appeared to control the inheritance of fruit yield per plant and average fruit weight. A × R crosses were comparable to B × R crosses in terms fruit yield and its contributing traits and had higher levels of anthracnose resistance to virulent isolate ‘Cc 38’. |
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Date |
2016-05-14T11:15:22Z
2016-05-14T11:15:22Z 2011-09-03 |
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Type |
Thesis
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Identifier |
Th-10111
http://krishikosh.egranth.ac.in/handle/1/65982 |
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Language |
en
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Format |
application/pdf
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Publisher |
University of Agricultural Sciences, Bengaluru
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