Reduction of oxalate levels in tomato fruit and consequent metabolic remodeling following overexpression of a fungal oxalate decarboxylase
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Title |
Reduction of oxalate levels in tomato fruit and consequent metabolic remodeling following overexpression of a fungal oxalate decarboxylase
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Creator |
Chakraborty, Niranjan
Ghosh, Rajgourab Ghosh, Sudip Narula, Kanika Tayal, Rajul Datta, Asis Chakraborty, Subhra |
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Description |
Accepted date: March 9, 2013
The plant metabolite oxalic acid is increasingly recognized as a food toxin with negative effects on human nutrition. Decarboxylative degradation of oxalic acid is catalyzed, in a substrate-specific reaction, by oxalate decarboxylase (OXDC), forming formic acid and carbon dioxide. Attempts to date to reduce oxalic acid levels and to understand the biological significance of OXDC in crop plants have met with little success. To investigate the role of OXDC and the metabolic consequences of oxalate down-regulation in a heterotrophic, oxalic acid-accumulating fruit, we generated transgenic tomato (Solanum lycopersicum) plants expressing an OXDC (FvOXDC) from the fungus Flammulina velutipes specifically in the fruit. These E8.2-OXDC fruit showed up to a 90% reduction in oxalate content, which correlated with concomitant increases in calcium, iron, and citrate. Expression of OXDC affected neither carbon dioxide assimilation rates nor resulted in any detectable morphological differences in the transgenic plants. Comparative proteomic analysis suggested that metabolic remodeling was associated with the decrease in oxalate content in transgenic fruit. Examination of the E8.2-OXDC fruit proteome revealed that OXDC-responsive proteins involved in metabolism and stress responses represented the most substantially up- and down-regulated categories, respectively, in the transgenic fruit, compared with those of wild-type plants. Collectively, our study provides insights into OXDC-regulated metabolic networks and may provide a widely applicable strategy for enhancing crop nutritional value. This work was supported by grants from the Indian Council of Medical Research (grant no. 63/2/2006–BMS) and the National Institute of Plant Genome Research, Government of India. R.G. and K.N. were supported by a predoctoral fellowship from the Council for Scientific and Industrial Research, Government of India. |
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Date |
2015-11-02T08:54:06Z
2015-11-02T08:54:06Z 2013 |
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Type |
Article
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Identifier |
Plant Physiol., 162(1): 364-378
0032-0889 http://www.plantphysiol.org/content/162/1/364.long http://172.16.0.77:8080/jspui/handle/123456789/299 |
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Language |
en_US
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Publisher |
American Society of Plant Biologists
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