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Ethylene acts as a negative regulator of glucose induced lateral root emergence in Arabidopsis

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Title Ethylene acts as a negative regulator of glucose induced lateral root emergence in Arabidopsis
 
Creator Singh, Manjul
Gupta, Aditi
Laxmi, Ashverya
 
Subject arabidopsis
auxin
brassinosteroid
ethylene
glucose
lateral root
 
Description Accepted date: 1 Jun 2015
Plants, being sessile organisms, are more exposed to the hazards of constantly changing environmental conditions globally. During the lifetime of a plant, the root system encounters various challenges such as obstacles, pathogens, high salinity, water logging, nutrient scarcity etc. The developmental plasticity of the root system provides brilliant adaptability to plants to counter the changes exerted by both external as well as internal cues and achieve an optimized growth status. Phytohormones are one of the major intrinsic factors regulating all aspects of plant growth and development both independently as well as through complex signal integrations at multiple levels. We have previously shown that glucose (Glc) and brassinosteroid (BR) signalings interact extensively to regulate lateral root (LR) development in Arabidopsis. (1) Auxin efflux as well as influx and downstream signaling components are also involved in Glc-BR regulation of LR emergence. Here, we provide evidence for involvement of ethylene signaling machinery downstream to Glc and BR in regulation of LR emergence.
This work was financially supported by the grant from
Department of Biotechnology, Ministry of Science and Technology, Government of India (BT/PR3302/AGR/02/814/2011),
NIPGR core grant and Council of Scientific and Industrial
Research, India (research fellowships to A.G. and M.S.).
 
Date 2016-01-22T09:48:03Z
2016-01-22T09:48:03Z
2015
 
Type Article
 
Identifier Plant Signal. Behav., 10(9): e1058460
1559-2324
http://172.16.0.77:8080/jspui/handle/123456789/580
http://www.tandfonline.com/doi/full/10.1080/15592324.2015.1058460
10.1080/15592324.2015.1058460
 
Language en_US
 
Publisher Taylor & Francis Group