Perioperative stress prolong post-surgical pain via miR-339-5p targeting oprm1 in the amygdala
Harvard Dataverse (Africa Rice Center, Bioversity International, CCAFS, CIAT, IFPRI, IRRI and WorldFish)
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Title |
Perioperative stress prolong post-surgical pain via miR-339-5p targeting oprm1 in the amygdala
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Identifier |
https://doi.org/10.7910/DVN/1V8QV2
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Creator |
Yi Zhu
Mei Sun Peng Liu Weidong Shao Ming Xiong Bo Xu |
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Publisher |
Harvard Dataverse
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Description |
The decreased expression of mu-opioid receptors (MOR) in the amygdala may be a key molecular in chronic post-surgical pain (CPSP). It is known that miR-339-5p expression in the amygdala of a stressed rat model was increased. Analyzed by RNAhybrid, miR-339-5p could target opioid receptor mu 1 (oprm1) which codes MOR directly. So, the authors hypothesized that miR-339-5p could regulate the expression of MOR via targeting oprm1 and cause the effects to CPSP. To simulate perioperative short-term stress, a perioperative stress prolongs incision-induced pain hypersensitivity without changing basal pain perception rat model was built. A pmiR-RB-REPORT™ dual luciferase assay was taken to verify whether miR-339-5p could act on oprm1 as a target. The serum glucocorticoid level of rats was test. Differential expressions of MOR, GFAP, and pERK1/2 in each group of the rats’ amygdala were tested, and the expressions of miR-339-5p in each group of rats’ amygdalas were also measured. |
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Subject |
Medicine, Health and Life Sciences
Amygdala Glucocorticoids MicroRNAs Models, Animal Pain Perception Pain, Postoperative Physiopathology Receptors, Opioid, mu Stress, Physiological |
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Contributor |
Nahm, Francis
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